Download Acute Pancreatitis: Research and Clinical Management by V. Becker (auth.), Hans G. Beger MD, FACS, Markus Büchler MD PDF

By V. Becker (auth.), Hans G. Beger MD, FACS, Markus Büchler MD (eds.)

Presents findings on acute pancreatitis within the fields of morphology, epidemiology, pathophysiology, enzymatic and in- strumental prognosis, scientific facets and pharmacological and surgical operation. Emphasis on medical implications at the neighborhood unencumber and vasoactice and poisonous components, the excessive price of bacterial an infection within the necroses, and the factors of impairment of cardiocirculatory, pulmonary and re- nal organs.

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Swollen mitochondria, dilated endoplasmic reticulum, and nuclear changes appeared within 30 min and progressed with time. Membrane and zymogen granule disintegration followed considerably later (3 - 6 h). This tissue disintegration took place in the absence of erythrocytes, PMNs and fibrin (since the necessary vascular supply was missing in vitro), so these elements, which are typical of acute pancreatitis, were not present. It appears that many of the changes observed in acute pancreatitis may be explained by ischemia, but that additional factors, including blood-borne elements and products of interaction between vascular and pancreatic elements, are necessary to produce the full picture.

Imrie CW, Benjamin IS, McKay AJ, Mackenzie L, O'Neill J, Blumgart LH (1978) A single centre, double-blind trial of Trasylol therapy in primary acute pancreatitis. Br J Surg 65:337-341 14. Ranson JHC, Spencer FC (1978) The role of peritoneal lavage in severe acute pancreatitis. Ann Surg 187:565-575 15. Madsen OG, Schmidt A (1979) Acute pancreatitis. A study of 122 patients with acute pancreatitis observed for 5-15 years. World J Surg 3:345-352 16. Kelly TR (1980) Gallstone pancreatitis: the timing of surgery.

4mmoIlI 46% 41% 49% 47% 58% 63% 61% 58% 43% 59% 28% 21% 27% 25% 23% 28% 31% 14% 17% 27% Cases 35 100 186 137 90 230 77 36 112 90 Total 1093 Percentage of increased triglycerides and cholesterol in diseases with high incidence of hyperIipoproteinemia. Table 3. 4 Chron. pancreatitis (n = 41) 20 17 60 3 Acute pancreatitis (n = 35) 7 12 72 9 Distribution (%) of types of hyperlipoproteinemia in unselected patients and in patients with chronic and acute pancreatitis. acute and 41 % of chronic pancreatitis.

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