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The Advances in Parasitology sequence includes in-depth experiences on present subject matters of curiosity in modern parasitology. It contains scientific experiences on parasites of significant impact, similar to trypanosomiasis and scabies, and extra conventional parts, corresponding to zoology, taxonomy, and existence heritage, which form present pondering and purposes. * sequence has the second one optimum ISI influence consider the parasitology team! (4.818 in 2002) * individuals are overseas specialists within the box
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Extra info for Advances in Parasitology, Vol. 58
ANTOINE ET AL. ILIL-10 IL-6 ILIL-4 ILIFNIFN-α/β ILIL-13 amastigotes NADPH NADP + H + + e- killed amastigotes H2O2 O2 apoptosis of infected MΦs TGFTGF-β O2OH- parasite molecules involved in the protection towards leishmanicidal mechanisms: LPG, PPG, GIPLs, trypanothione, SOD, catalase Fas FasL L-arginine + O2 effector T lymphocyte CD40L NO RNI ? non replicative amastigotes NO synthase 2 NO CD40 NADPH oxidase activation inhibition MIF TNFTNF-α ILIL-7 IFNIFN-α/β IFN-γ Figure 6 Mechanisms of Leishmania killing developed by MÈs and how parasites can evade them.
The amino acid L-ornithine is an essential compound for biosynthesis of the polyamines spermidine and putrescine. , 2001). As polyamines are key components for parasite proliferation, MÈs activated in that manner are highly permissive for parasite expansion. LEISHMANIA SPP. , 2001; Sacks and Sher, 2002). There is no doubt from in vitro experiments that DCs can phagocytose Leishmania but whether both parasite stages can be efficiently phagocytosed by DCs is still a debated question. , 2004). Likewise, results bearing on the parasite survival in DCs show great variability.
1. The Different Pathways Leading to the Development of Leishmanicidal Properties Despite the fact that parasites have developed strategies to down regulate signalling pathways leading to enhancement of MÈ leishmanicidal properties, it is well known that in L. major-resistant mice, which control the parasite multiplication after a short period of parasite amplification, MÈs acquire an activated microbicidal LEISHMANIA SPP. 33 phenotype. How this phenotype is acquired during Leishmania infection is, however, not very well understood.